Atherosclerosis

Atherosclerosis like any disease comes about as a malfunction of one or more of your bodies systems.  Much if not most of the time this is a result of a slow degenerative process due to the lack of adequate bodily supplies of the elements necessary for normal function and rejuvenation of affected organs. Commercial Farming and natural erosion has depleted global farmlands of most essential elements therefore it is not wise to assume that your diet contains enough of these elements for normal body function and maintenance. See Senate Document 264. It is also unwise to suspect that intervention therapies will be successful without adequate dietary levels of these elements in useable forms.             See The Package. Does what you eat provide all the essential elements you need? See Senate Document 264. Just how good is the "best" diet?  See The Food Pyramid

ATHEROSCLEROSIS is a disease that can affect people at any age, although it usually doesn't pose a threat until people reach their forties or fifties. It is characterized by a narrowing of the arteries caused by cholesterol-rich plaques of immune-system cells. Key risk factors for atherosclerosis, which can be genetic and/or environmental, include: elevated levels of cholesterol and triglyceride in the blood, high blood pressure and cigarette smoke.
  A protein called apolipoprotein E, which can exist in several different forms, is coded for by a gene found on chromosome 19. It is important for removing excess cholesterol from the blood, and does so by carrying cholesterol to receptors on the surface of liver cells. Defects in apolipoprotein E sometimes result in its inability to bind to the receptors, which leads to an increase a person's blood cholesterol, and consequently their risk of atherosclerosis.
  Currently, a debate is raging over how the various mutated forms of apolipoprotein E effect the body. As a result, many of the treatments proposed remain in their experimental phase. While mice are proving useful for modeling the human disease, a great deal of research is still required before we can fully understand the mechanisms that regulate the levels of lipoproteins - like apolipoprotein E - in the blood.